Search results for "HOG pathway"

showing 3 items of 3 documents

Histidine kinases mediate differentiation, stress response, and pathogenicity in Magnaporthe oryzae.

2014

The aim of this study is a functional characterization of 10 putative histidine kinases (HIKs)-encoding genes in the phytopathogenic fungus Magnaporthe oryzae. Two HIKs were found to be required for pathogenicity in the fungus. It was found that the mutant strains ΔMohik5 and ΔMohik8 show abnormal conidial morphology and furthermore ΔMohik5 is unable to form appressoria. Both HIKs MoHik5p and MoHik8p appear to be essential for pathogenicity since the mutants fail to infect rice plants. MoSln1p and MoHik1p were previously reported to be components of the HOG pathway in M. oryzae. The ΔMosln1 mutant is more susceptible to salt stress compared to ΔMohik1, whereas ΔMohik1 appears to be stronger…

MagnaportheHistidine KinaseMutantVirulenceconidiaBiologyMicrobiologyMicrobiologyFungal Proteinshypoxia signalingGene Expression Regulation FungalpathogenicityAppressoriaPlant DiseasesOriginal ResearchAppressoriumFungal proteinVirulenceHistidine kinaseHOG pathwayOryzadifferentiationMagnaporthe oryzaeSpores Fungalbiology.organism_classificationYeastMagnaportheMultigene FamilyPhosphorylationProtein KinasesMicrobiologyOpen
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Rapid adaptation of signaling networks in the fungal pathogen Magnaporthe oryzae

2019

Abstract Background One fundamental question in biology is how the evolution of eukaryotic signaling networks has taken place. “Loss of function” (lof) mutants from components of the high osmolarity glycerol (HOG) signaling pathway in the filamentous fungus Magnaporthe oryzae are viable, but impaired in osmoregulation. Results After long-term cultivation upon high osmolarity, stable individuals with reestablished osmoregulation capacity arise independently from each of the mutants with inactivated HOG pathway. This phenomenon is extremely reproducible and occurs only in osmosensitive mutants related to the HOG pathway – not in other osmosensitive Magnaporthe mutants. The major compatible so…

GlycerolMagnaportheved/biology.organism_classification_rank.speciesMutantGenomeSalt StressTranscriptome0302 clinical medicineOsmoregulationLoss of Function MutationGene Expression Regulation FungalGene Regulatory NetworksSuppressorReestablishment of osmoregulation0303 health sciencesbiologyMagnaporthe oryzaeRewiringAdaptation PhysiologicalRapid adaptationCell biologyMagnaportheOsmoregulationEpigeneticsGenome FungalBiotechnologySignal TransductionResearch Articlelcsh:QH426-470lcsh:BiotechnologyDioxolesFungal Proteins03 medical and health sciencesDrug Resistance Fungallcsh:TP248.13-248.65GeneticsPyrrolesModel organismGene030304 developmental biologyPlant DiseasesOsmotic concentrationved/biologyGene Expression ProfilingEvolution of signaling networksHOG pathwayOryzabiology.organism_classificationlcsh:Genetics030217 neurology & neurosurgery
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Sonidegib en el tratamiento del carcinoma basocelular localmente avanzado

2021

Resumen: Sonidegib es un inhibidor del receptor transmembrana Smoothened (SMO), de la vía de señalización de Hedgehog, indicado para el tratamiento del carcinoma basocelular localmente avanzado (CBCla), no susceptible a cirugía curativa ni a radioterapia. Sonidegib ha demostrado su eficacia y seguridad en pacientes con CBCla en el ensayo de fase II (BOLT), donde el 61% (IC 95%: 48; 72) de los pacientes tratados con 200 mg de sonidegib tuvo una respuesta objetiva al tratamiento, con un tiempo medio hasta la respuesta de cuatro meses. La mediana de duración de respuesta fue de 26,1 meses y la mediana de supervivencia libre de progresión fue de 22,1 meses. Los eventos adversos más frecuentes f…

Hedgehog pathway inhibitorsHedgehog signaling pathwayRL1-803General MedicineDermatologyLocally advanced basal cell carcinomaInternal medicineRC31-1245SonidegibActas Dermo-Sifiliográficas
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